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Reconsideration of amyloid hypothesis and tau hypothesis in Alzheimers disease

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Paradoxically, although glial-derived S100 promotes neuronal remodeling through Cx43-dependent mechanisms under homeostatic condition, its overexpression during intestinal inflammation triggers widespread glial activation, thereby exacerbating neuroinflammation and neuronal damage (31)

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The drop in leukocyte counts is particularly significant, as it points to reduced systemic inflammation

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