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doi: 10.3389/fpsyt.2020.595584 PMID: 33424664 Gessa GL et al (1963): On the mechanism of hypotensive effects of MAO inhibitors

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identifies HIF1a and RIPK3 as critical players for the control of intestinal epithelial cell survival during the course of CIA [1]

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423 The research further demonstrated that this anti-inflammatory effect was not mediated by GLP-1R in the blood or endothelial cells but required the GLP-1R in the CNS

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