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These structures suggest that Phe443 and Asp538 at the S2 site are key residues in SAM binding (Fig

thymosin 4 structure, function and the early clinical programme for the full protein (distinct from the TB-500 fragment)

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Therefore, the conclusions drawn from this review are limited by the quality and quantity of the studies included in the analysis
Aging 34 , 15811588 (2013)
