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Striatal oxidative damage parallels the expression of a neurological phenotype in mice transgenic for the mutation of Huntington's disease

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Although modest in scale and performed without MRI-guided targeting, the study offers compelling evidence that restoring cerebellar-cortical communication can translate into meaningful functional gains

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Fahr syndrome stems from secondary causes such as endocrine or mitochondrial issues (3) Methods: This retrospective study reviewed patients with Fahr's disease or syndrome diagnosed between 2018 and 2024, based on neuroimaging showing bilateral basal ganglia calcifications