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10.1186/s13048-022-01080-3 32 ChiY

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Our results further show that efficient conversion depends on a defined balance between free thiolate and nitrosylated cysteine species, consistent with a mechanism in which transient S-nitrosylation facilitates thioldisulfide exchange

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JAK/STAT3 pathway is involved in survival of neurons in response to insulin-like growth factor and negatively regulated by suppressor of cytokine signaling-3