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Simultaneously, Fe2+ oxidizes lipids via the Fenton reaction, leading to ROS buildup and the progression of ferroptosis [15]

The patient was initiated with Intravenous Immunoglobulin (IVIG) therapy

Neutralizing antibodies to IL-18 ameliorate experimental autoimmune neuritis by counter-regulation of autoreactive Th1 responses to peripheral myelin antigen

Furthermore, primary antioxidants interact with the oxidants (free radicals) to transform them into more stable, non-reactive products

Spatiotemporally separable Shh domains in the midbrain define distinct dopaminergic progenitor pools