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Advanced technologies for differentiating kidney organoids from hPSCs and efficient genome-editing systems with the clustered regularly interspaced short palindromic repeat CRISPR-Cas9 have enabled modeling of human kidney diseases 15,16,17

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Even if the specimens are handled properly, it can be challenging to detect even one mediator elevated because of the timing of mast cell degranulation

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Consistent with our FSP1 GFP-P2A-BFP cells, loss of RFK and FLAD1 resulted in lower FSP1GFP fluorescence in cells expressing ectopic WT FSP1GFP (Fig

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