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ROS and RNS may trigger programmed cell death, e.g., apoptosis [29], necessary for tissue remodeling during the development and normal tissue turnover [30], proliferation, differentiation, maturation of neural stem cells, dendritic processes, and synaptic plasticity [31,32,33]

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These findings were further expanded upon by Regan et al who was able to recreate the SOF+amiodarone induced bradycardia in animal models using guinea pigs and rhesus monkeys[94]

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