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The NLRP3 inflammasome is activated by various danger signals, including PAMPs, DAMPs, and metabolites associated with metabolic disturbances (such as ATP and K + )

This improved vascularization creates a more favorable environment for the migration and proliferation of fibroblasts and other cell types essential for wound healing

In the absence of a unified conceptual framework, distinctions between neuroprotection, symptomatic treatment, and disease modification often become obscured, risking the conflation of mechanistic plausibility with demonstrable clinical benefit

G., Yolken, R

BNIP3 interacting with LC3 triggers excessive mitophagy in delayed neuronal death in stroke

Harnessing the power of bee venom for therapeutic and regenerative medical applications: an updated review