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Ferroptosis was first defined [4] as a distinct form of regulated cell death induced by erastin

28,29,30 NOX-mediated ROS broadly and specifically regulate various redox-sensitive signaling pathways involved in cancer progression via stimulating oncogenes, such as Src and Ras, and inactivating tumor suppressor proteins, such as TP53 and PTEN

Sphingosine-1-phosphate promotes the proliferation and attenuates apoptosis of Endothelial progenitor cells via S1PR1/S1PR3/PI3K/Akt pathway

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doi: 10.3945/an.116.013912