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According to the work of Stamler et al, (1993), homocysteine auto-oxidation might prevent thiol nitrosation that, under normal physiologic conditions, sustains the antiplatelet and vasorelaxing activities of NO by increasing the stability of NO (Simon et al, 1993

This interaction appears to initiate a series of intracellular signaling cascades

Other established risk factors include: Hyperlipidemia Metabolic syndrome Obesity Atrial fibrillation Chronic kidney disease Sedentary lifestyle Sleep disorders, particularly obstructive sleep apnea Sleep disorders may contribute to CSVD through intermittent hypoxia, sympathetic activation, endothelial dysfunction, and impaired glymphatic clearance

doi: 10.1007/s12028-010-9445-z [PubMed] 21967361 Silbergleit R, Lowenstein D, Durkalski V, Conwit R

Dose and administration Deep intramuscular injection: - Piglets: 1 - 1.5 ml per piglet at 3 - 5 days of age, repeat after 7 days

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