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Considering tumors as relatively homogeneous entities exclusively composed of malignant cells and attributing CDDP resistance only to cancer cell-intrinsic mechanisms (hence neglecting the prominent structural and functional impact of stromal and immune cells) appears indeed as an excessive oversimplification (see below), and may in part explain why clinically applicable strategies for restoring CDDP sensitivity are still missing

Improvement of key molecules in glucose metabolism, fat metabolism, and amino acid metabolism (e.g., glucose6phosphate dehydrogenase, triglycerides, and levodopa) might be potentially beneficial in PD

10.1159/000520726 62 SunY.YangY.LiuS.YangS.ChenC.LinM.et al (2022)

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By unregulating Notch and p53 signaling pathways and secreting matrix metalloproteinases (MMPs), growth factors, and cytokines, they stimulate angiogenesis, inflammatory response, cell proliferation, and invasion (67, 68)

Whereas the binding of CD28 to B7 is costimulatory, the binding of CTLA-4, which is more avid than that of CD28, is coinhibitory