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Depletion of GSH due to intensified oxidative stress or the action of inhibitors significantly reduces the ability of GPX4 to counteract lipid peroxidation, leading to increased lipid peroxides and subsequent ferroptosis (151)

There are, however, several important considerations that must be recognized by both the physician and patient

HBV infection is able to change the levels of antioxidant enzymes in an Nrf2-/antioxidant response element (ARE)-dependent and -independent fashion [115]

From Blastp analysis within Burkholderia , only B

a tissue specific promoter is only active in specific types of differentiated cells/tissues