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Early after liver resection, regeneration ability is insufficient due to the shortage of liver cells and liver volume, high pressure in the portal vein, long sustained time and small liver syndrome that is characterized by postoperative liver dysfunction, high blood bilirubin with prolonged cholestasis, blood coagulation disorders, portal hypertension, and the emergence of serious ascites

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The gold standard for diagnosis is LCMS/MS as part of NBS, and genetic testing is sometimes recommended to identify the specific mutation causing the condition

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Alix-mediated assembly of the actomyosin-tight junction polarity complex preserves epithelial polarity and epithelial barrier

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