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Recently, growing evidence supports the concept that AD is a metabolic disease of type 3 diabetes mellitus, based on the fact that insulin resistance and dysfunctional glucose metabolism in the brain are often found in patients with AD and that A deposition induces reduced expression of insulin receptor on neuronal cells (

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Selenium deficiency impairs the glutathione antioxidant system even when glutathione levels are adequate [26]

Consistent with this idea, in rodents, brain glycogen levels are decreased after periods of intense neuronal activity such as during sensory stimulation (Swanson et al