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This interplay contributes to neuronal dysfunction and apoptosis in neurodegenerative diseases like AD, and cardiomyocyte apoptosis in cardiovascular diseases [72, 73]

The overactivation induced by a synergistic effect with estrogen may partly explain the detrimental impact of excessive androgen on mitochondria in females but needs further validation

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& Feldman, E

At the initial baseline visit, serial venous sampling performed after oral d 3 -l-carnitine challenge revealed rapid increases in plasma concentrations of d 3 -l-carnitine, and subsequent increases in d 3 -BB and d 3 -TMAO following a lag phase (Figure 1, AC, filled circles)

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