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The PMRS transfers electrons from the intracellular cytosolic RBC compartment to the extracellular medium by oxidizing intracellular electron donors like ascorbate and NADH, acting as a compensatory mechanism against elevated ROS/OS, and may contribute to redox regulation throughout the body [42]

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Oxidative inflammation is also thought to be a major cause of molecular damage through various stressors (McGarry et al., 2018)

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Proteasome inhibition suppresses essential immune functions of human CD4+ T cells