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Given that SIRT7 is a deacetylase, we hypothesized that SRF may undergo SIRT7-mediated deacetylation to modulate VSMC phenotypic switching 29
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doi: 10.1093/annonc/mdl105
USA 102 , 1060410609 (2005)
Similar content being viewed by others Background The mesenchymal-epithelial transition (MET) can be utilized as a signaling receptor for hepatocyte growth factor (HGF), and it has been reported that in humans, the MET proto-oncogene encodes for the receptor tyrosine kinase (RTK) of growth factor tyrosine kinase [1, 2]
Human milk contains antioxidant enzymes such as SOD, CAT, and GPX, which are crucial for preventing lung damage in neonates
