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Increased plasma GSH led to cysteine depletion, impaired protein synthesis, decreased GPx, and increased GST activities [134]

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Hydration of the C2 C3 double bond results in the formation of AFB2a which is rapidly formed in certain avian species [45]

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Alterations in the normal function of excitatory amino acid carrier 1 affect cysteine transport and GSH synthesis impairing zinc homeostasis (the thiol group of GSH can function as a principal Zn 2+ chelator for the maintenance of Zn 2+ homeostasis in neurons) and oxidative stress, enhancing susceptibility to ischemia-induced neuronal cell death in the hippocampus (162164)