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Recent studies have revealed that precancerous lesions of HGSOC are primarily located in the fallopian tube epithelium and are driven by TP53 mutations (7,26,54)

HMGCS2 silencing attenuates high glucose-induced in vitro diabetic cardiomyopathy by increasing cell viability, and inhibiting apoptosis, inflammation, and oxidative stress

Their stability in circulation makes them promising candidates for clinical applications

Thioredoxin reductase activity is decreased by selenium deficiency

As age-related cataract progression in humans is very slow and therapeutics must be applied for a long duration to ameliorate or prevent the development of cataracts, it is vital to study the long-term safety of pharmacological therapies