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Lysosomal cholesterol accumulation in macrophages leading to coronary atherosclerosis in CD38(/) mice

The present study, for the first time, demonstrates an underlying mechanism for the activation of eNOS by BPC 157 on stimulating the Cav-1 phosphorylation and reducing the binding of eNOS to Cav-1

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Cells treated with AHK-Cu proliferated faster than untreated cells

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