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Mitochondria can release Ca 2+ into the cytosol via the mitochondrial permeability transition pore (mPTP) or the Na + /Ca 2+ exchanger [23, 24]

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Histological findings from both animal models and human studies have described sinusoidal dilatation, microvesicular steatosis, and pericentral necrosis, changes commonly associated with oxidative and inflammatory stress

Statistical Analysis A two-tailed p 0.05)

It is essential for the initial transport of activated long-chain fatty acids across the mitochondrial membrane via the CPT system, thus governing the substrate flux available for both -oxidation and subsequent ketogenesis

Considering the amount of total carnitine content in the body (about 20 g) [9], the low carnitine bioavailability and the large amount of carnitine lost with the urine after integration, a very high dose of carnitine and for long periods should be integrated to obtain a real increase in the amount of muscular carnitine in healthy subjects