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Cotranslational proteolysis dominates glutathione homeostasis for proper growth and development

References 1.Nagai S, Black S

You may have heard it called fatty liver disease, but that name is no longer being used in the medical community

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Chondrocytes that lack G6PDH are NADPH-insufficient and, in response to the impaired GSSG recycling and reduced protein folding, they activate the UPR and protein degradation to avoid proteotoxicity and cell death

55 These potential therapeutics targeted towards rejuvenating, optimizing, and recruiting endogenous stem cells will likely increase the efficacy of cartilage tissue engineering techniques in the older patient population