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Interestingly, undifferentiated NSCs rely on glycolysis, and during the differentiation process, metabolism switches towards OXPHOS (Mandal et al., 2011

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Accordingly, in IRI mouse model, simultaneous blockade of LPA1 and LPA3 receptors improves renal function, and the protective effect of the dual antagonist is reversed after the use of OMPT, an LPA3 agonist, suggesting that LPAR3 activation may promote renal injury in IRI [62]

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