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Mechanisms: Its ability to support microvascular integrity, enhance fibroblast migration, and reduce oxidative stress facilitates recovery across muscle, tendon, ligament, nerve, gastrointestinal lining, and skin

In animal models, BPC-157 improved healing in tendons and ligaments, including enhanced MCL repair and accelerated Achilles tendon recovery (Cerovecki et al., J Orthop Res, 2010)

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Daniel Drucker's research at the Lunenfeld-Tanenbaum Research Institute at Sinai Health in Toronto helped pave the way for GLP-1 medications

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