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Cell death induced by cisplatin is concentration dependent and includes necrosis and apoptosis mechanisms (Gonzalez et al., Cisplatin distorts the structure of the DNA that generate intrastrand 1, 2crosslinks binding proteins into shallow minor groove [high-mobility group (HMG) box proteins, repair proteins, transcription factors, histone H1] (Kartalou and Essigmann, 2002

Addition of Gemtuzumab Ozogamicin (GO) to Fludarabine, Cytarabine and G-CSF (FLAG) Based Induction Regimen Results in Better Early Molecular Response and Relapse Free Survival Compared to Idarubicin (FLAG-Ida) in Newly Diagnosed Core Binding Factor Leukemia

Tamouza H, Vende F, Tiwari M, Arcos-Fajardo M, Vrtovsnik F, Benhamou M, et al

Wang et al., 2025

Werte, die das Ausmass des oxidativen Stresses anzeigten sowie Werte, die das Immunsystem beurteilen helfen (Aktivitt der Natrlichen Killerzellen und auch anderer Abwehrzellen (B-Lymphozyten)), besserten sich allesamt genauso wie das Verhltnis von oxidiertem zu reduziertem Glutathion

Studies revealed that by increasing the concentrations of the cytosolic cysteine, and thus glutathione of monocytes and macrophages using a synthetic form of cysteine called NAC, blocks H 2 O 2 mediated activation of NF-kB, and thus pro-inflammatory cytokines (tumor necrosis factors [TNF]-, interleukin [IL]-1 and IL-6) by this route