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The condition is caused by a cytoskeletal protein deficiency in the RBC membrane

In L02 cells overexpressed with FABP1, the total intracellular FABP1 proteins gradually declined in response to treatment with CHX, a protein synthesis inhibitor [35], whereas overexpression of GSTA1 led to an acceleration of the decline with a shortening of the half-life of FABP1 (Figure 7E)

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doi: 10.15761/CMI.1000103 38

Calcif Tissue Int, 1997

An olive-derived extract 20% rich in hydroxytyrosol prevents -amyloid aggregation and oxidative stress, two features of Alzheimer disease, via SKN-1/NRF2 and HSP-16.2 in Caenorhabditis elegans