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All the three indexes showed a significant decrease after theaflavins intervention, with the effect of the theaflavins-treated group being more pronounced in the HO-1 knockdown cells compared to normal cells (Fig
Residue Pro39 also interacts with nucleotide bases (at consensus positions + 3 and + 8) via additional van der Waals interactions
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doi:10.1016/j.mehy.2009.12.018
TNF-mediated necroptosis in brain endothelial cells as a potential mechanism of increased seizure susceptibility in mice following systemic inflammation
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