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The primary function of IDH1 and IDH2 is to serve as producers of NADPH to enhance the cellular responses to oxidative stress and the generation of reactive oxygen species, ROS

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However, emerging research indicates that c-Rel may serve as a potential therapeutic checkpoint in MDSCs, selectively promoting pro-tumoral gene expression while repressing anti-tumoral genes via a c-Rel enhanceosome complex [87]

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These findings have led to the hypothesis that DSIP may play a role in influencing circadian and sleep-wake regulatory mechanisms in non-human research subjects in preclinical trials