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Its cellular expression is controlled at the transcriptional level by nuclear factor (erythroid-derived 2)-related factor 2 (NRF2) (52), and at the post-transcriptional level, through degradation and deadenylation/polyadenylation processes (53)

DP downregulates CINC-3 expression Considering the pivotal role of CINC-3 in neutrophil recruitment during inflammatory responses, We further detected the expression levels of CINC-3 in the control and DP groups using the ELISA method.The results indicated that, on the first day post-DP treatment, the expression level of CINC-3 was decreased compared to that in the control group

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In biochemical and cellular research models, Epithalon has been examined for its interactions with nuclear and chromatin-associated processes, including modulation of gene expression and telomere-related regulatory mechanisms

Chemotherapy Agents Many chemotherapy drugs rely on oxidative stress to destroy cancer cells