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[DOI] [PubMed] [Google Scholar] 185.Kopelman P, Bryson A, Hickling R, et al

doi: 10.1186/1471-2350-8-S1-S13

doi: 10.1016/j.chemosphere.2014.10.039 192 MuozXBarreiroEBustamanteVLopez-CamposJLGonzlez-BarcalaFJCruzMJ

Since necrosis can activate an immediate immune response during ischemic injury, the induction of an inflammatory reaction and the release of free radicals lead to increased oxidative stress, cell death, and organ damage [12,13,14,15,16,17,18,19,20,21,22,23,24,25,30,33,34,35,36,37,38]

Front Cell Infect Microbiol (2017) 7:276

Initial analysis of our RNA-seq dataset comparing melatonin- and vehicle-treated ILC2s revealed broad suppression of pro-inflammatory gene expression following melatonin treatment, including canonical type 2 cytokines such as Il5 , Il13 , and Csf2