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Studies on mice deficient in CR1/2 or C3 (complement proteins) revealed a reduced ability to mount a humoral response to AAV compared to mice previously infected with wild-type AAV, emphasizing the complement systems role in generating AAV-specific antibodies [104]

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Under steady-state conditions, even during acute lung inflammation, the efficient cellular clearance of alveolar macrophages results in a minimal presence of apoptotic cells in the airways

The chemical reaction involves the deprotonation of C4 in the A ring of the steroid substrate, thereby enabling the double bond between C5 and C6 to isomerize to a double bond between C4 and C5 (Figure 2)