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In metabolomics, significant alterations were observed in metabolism related to arginine and proline, arachidonic acid, glutathione metabolism, and ubiquinone and other terpenoid-quinone biosynthesis pathways as CKD progressed with further metabolomic analyses revealing that variations in the distributions of pro-oxidant and toxic metabolites from the four identified metabolic pathways were observed in the feces and serum of the patients as the disease progressed

doi: 10.1038/s41577-019-0210-z

and (3) Gene therapy approaches that correct the defective lysosomal cystine transport in parenchymal cells

The rest of the procedures are as follows: 1

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