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In neuroendocrine prostate tumors, loss of tumor suppressor protein kinase C (PKC) / enhanced serine metabolism through mTORC1/ATF4/PHGDH axis, which leads to aberrant DNA methylation by increased one-carbon metabolic flux, ultimately leading to treatment resistance and poor prognosis [98]

However, the instability of the bacterial host protease system and associated plasmid loss can result in a decrease in recombinant lipase production

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The activity of lipase is a utility of interfacial conformation: the enzyme can be denatured as well as triggered or neutralized and the interface is a suitable spot for restraining lipolysis

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