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The restoration of mTOR signaling with Fer1 rescue treatment in the context of RSL3 effects, and the genetic knockdown of GPX4, reaffirm our in vitro findings that RSL3-mediated induction of ferroptosis, mTOR pathway suppression, DNA damage and failure of DNA repair, and autophagy are GPX4-dependent processes in thyroid cancer cells
Bernards, High-throughput functional genetic and compound screens identify targets for Senescence induction in Cancer
10.1095/biolreprod58.1.261 Biol
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