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The absence of human teratogenicity data means risk cannot be quantified, but early first-trimester exposure coincides with organogenesis when developmental signaling is most vulnerable to external modulators

[DOI] [PMC free article] [PubMed] [Google Scholar] 119.Sen G.L., Webster D.E., Barragan D.I., Chang H.Y., Khavari P.A

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Techniques such as allotopic expression and gene editing hold great promise for future treatments

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