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Whereas cardiomyocyte hyperplasia brought about by increased microcirculatory blood supply (e.g., during pregnancy, growth, or when undergoing athletic training) is considered physiological and perfectly reversible, adverse or pathological remodeling following myocardial infarction (MI) is associated with disproportionate risk for heart failure (HF) and carries significant worse survival [42]

Figure 5A, B show obvious heterogeneity distribution of the fluorescence signal in the cornea

Escherichia coli mutants deficient in the aspartate and aromatic amino acid aminotransferases

This is an important limitation of our study