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The mechanism involves glycine exerting a hyperpolarizing effect on macrophages and other immune cells, inhibiting calcium influx through voltage-sensitive channels and thereby dampening their inflammatory activity
To view a copy of this licence, visit About this article Cite this article Dbrowski, G., Konopka, I
Long exposure to high glucose concentration impairs the responsive expression of g-glutamylcysteine synthetase by interleukin-1b and tumor necrosis factor-a in mouse endothelial cells
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A Shortage of FTH induces ROS and sensitizes RAS-proficient neuroblastoma N2A cells to ferroptosis
