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Central regulators of mitochondrial reprogramming: coordinated control by metabolic enzymes, spatiotemporal heterogeneity, and post-transcriptional networks Building on previous sections that examined how CRC develops resistance through mitochondrial metabolic reprogramming, this section explores three key regulatory mechanisms that sustain such adaptability

Neuroprotective Action

Sodium butyrate inhibits high cholesterol-induced neuronal amyloidogenesis by modulating NRF2 stabilization-mediated ROS levels: involvement of NOX2 and SOD1
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Intriguingly, both Sertoli cells and germ cells express L-CPT I and M-CPT I, implying that carnitine might travel from the bloodstream to Sertoli cells and germ cells

N-acetyl-L-cysteine suppresses TGF-beta signaling at distinct molecular steps: the biochemical and biological efficacy of a multifunctional, antifibrotic drug
