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In plain terms, while the raw percentage increases varied, the variation could easily be explained by chance rather than a true superiority of one method over another
Thus, TB-500 may be hypothesized to support angiogenesis by combining a cytoskeleton-linked increase in endothelial motility with signaling changes that raise pro-angiogenic programs (VEGF-A and Ang2/Tie2) via Notch/NF-B coupling in damaged cellular structure
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10.1038/srep01814 25 FrancisS
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