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While our study focused on endothelial K-Hcy-HMGB1/2-mediated senescence, K-Hcy modifications may constitute a novel pathological axis that disrupts multiple proteins and cellular processes, thereby driving ED through broader and previously unrecognized mechanisms
hepatocyte-specific SIRT1 deletion primarily aggravates metabolic dysfunction
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Stem cell factor SOX2 confers ferroptosis resistance in lung cancer via upregulation of SLC7A11
Lancet 365 , 15521560 (2005)
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