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4.1 Pharmacokinetic and pharmacodynamic considerations of antioxidant supplementation in AMS As mentioned above, oxidative stress has been proposed as a key contributor to the pathophysiology of AMS, driven by hypobaric hypoxiainduced mitochondrial dysfunction, increased ROS generation, endothelial activation, and neurovascular dysregulation

Accessed 11 June 2021 Grady MM, Stahl SM

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SIRS are nonspecific and may or may not be related to infections by pathogens and is defined by the following clinical criteria: fever greater than 38 C or less than 36 C

They all have a flavin-containing reductase subunit that uses NADPH, and not NADH, as a substrate

Liss, 231-252, 1990