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Therefore, the metabolic pathways of glutamine, tryptophan, and leucine converge on key regulatory nodes such as mTOR, AhR, and epigenetic modifiers, creating a network that dynamically controls Th17/Treg equilibrium

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It is notable that treatments that favor ER oxidation such as diamide or overexpression of constitutively active ERO1 elicit only modest ER stress (Hansen et al., 2012) (data not shown)

High-fat diets disrupt brain lipid metabolism and promote neuroinflammation, increasing depression risk (Wang A

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