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therefore, that oxidative stress in lymphoid cells may impair the antiviral immune response, thus facilitating chronicity of HCV infection [1]

This evolution reflects GPxs growing recognition in preventive medicine and personalized health strategies

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Among them, the STGE motif contained in p62 can exhibit a high affinity for Keap1 after phosphorylation by signaling kinases such as casein kinase 1 (CK1), transforming growth factor -activated kinase 1 (TAK1), mTORC1 and PKC similar to the ETGE motif of Nrf2 (Ichimura et al

, Rodriguez, F., Lherbet, C.**, Mourey, L.** (2025) Is Mycobacterial InhA a Suitable Target for Rational Drug Design

doi: 10.1038/s41392-020-00216-5