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Furthermore, interactions of brain interstitial A with microglia [87, 88] and exposure to transition metals [81,82,83] also contribute to oxidative stress in neurons

This regulatory axis is not merely theoretical, but it has been consistently demonstrated using natural products that activate the NRF2-ARE pathway

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The modification of lipoproteins, specifically LDL and HDL, by MG further fuels the flames of atherosclerosis

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The increase in esterified AX suggests that an early enzymatic mechanism leading to the acylation reaction occurs during embryogenesis [90]