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This mutation caused a defective UGT1A1 gene that cannot correctly produce a necessary enzyme described above, called uridine diphosphate glucuronosyltransferase (UGT)

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5 However, its most important antioxidant effect is thought to be as a precursor for glutathione (GSH), 6 a tripeptide made from the amino acids glutamate, glycine and cysteine, and the bodys major antioxidant

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