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Instead of being burned for fuel, these fats accumulate outside the mitochondria, leading to lipotoxicity and further cellular stress

Because GBM has evolved a range of abnormal metabolic pathways in response to the changing TME, it is possible to design ferroptosis-related core molecular inducers to reverse these metabolic processes

"Genome Instability in Development and Aging: Insights from Nucleotide Excision Repair in Humans, Mice, and Worms"

XuYLvLLiuYSmithMLiWTanXet alTumor suppressor TET2 promotes cancer immunity and immunotherapy efficacy

10.1016/j.urology.2003.10.034 [DOI] [PubMed] [Google Scholar] Chen L., He P

3.3 Assembly, function enrichment analysis and identification of differentially expressed genes In this study, the H-0 group was used as the control, and the number of differential genes in H-96a, H-96b and H-96c groups were counted ( Figures 4 , 5 )