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It has been studied for its capacity to restore thymic function, regulate T-cell differentiation, and support cellular immunity in aging and stress-related research contexts.

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Mitochondrial dysfunction is increasingly recognized as a contributor to neuropathy, particularly in diabetic and chemotherapy-induced forms where mitochondrial damage in dorsal root ganglion neurons precedes and contributes to axonal degeneration

Results were not statistically significant for weight loss compared to placebo
Mechanism of oxidative stress in neurodegeneration
