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[DOI] [PMC free article] [PubMed] [Google Scholar] 230.Schuetz YB, Naik A, Guy RH, Kalia YN

These observations strongly implicate microglia in disease progression, though whether their activation primarily drives pathology or represents a protective response remains debated

Nature 392 , 615618 (1998)

Within HSCs, curcumin enhances PPAR functionality while mitigating oxidative stress through Ob-R dephosphorylation, subsequently suppressing Ob-R transcription and blocking leptin-mediated signaling, thereby abolishing leptins pro-activation effects on HSCs (Tang et al., 2009)

Moreover, despite their biomimetic architectural excellence, these systems inevitably constitute simplified approximations of the extraordinarily complex in vivo physiological milieu, potentially obscuring critical intercellular communication networks, systemic immunological responses, and multi-organ interaction paradigms that characterize authentic pathophysiological processes

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