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Expression of GPX4 regulates ROS-induced cell death in LCL-K cell Based on the finding that GPX4 overexpression in DLBCL correlated with poor patient prognosis and negatively correlated with intracellular ROS accumulation as determined by 8-OHdG staining, we hypothesized that overexpression induces resistance of tumor cells to ROS-induced cell death

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NK-EVs exert antitumor effects by enhancing the activation of specific apoptotic markers in hepatocellular carcinoma cells (HCC) (Hep3B) through the inhibition of serine/threonine protein kinase phosphorylation (Fig

Is CFTR an exchanger?: Regulation of HCO 3 transport and extracellular pH by CFTR